Leakage of fibrinogen and coagulation factors through the damaged endothelium causes fibrin deposition in vessel walls, which appear as circumferential, acellular eosinophilic deposits (fibrinoid necrosis).
Over time, release of growth factors by damaged tissue stimulates the formation of concentric layers of collagen and proliferating smooth muscle cells, resulting in an☆ "onion skin" appearance (hyperplastic arteriosclerosis) of the arteriole.
affected kidney→ ☆mild atrophy
UA→ bla...
renal
cardiology
UWorld
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