SVT treatment(supraventricular tachycardia)
β blocker, Ca blocker(blocks AV node),adenosine
+vagal maneuver,cardioversion,ablation
Ca blocker is contraindicated with pseudoVT(WPW+AF),retrograde AVRT
(when AV node is blocked, retrograde AVRT will be aggravated) cardiology HirotoShishido
more common papillary muscle ruptureposteromedial papillary muscle rupture
(because it has a single blood supply from PDA(posterior descending artery))
→MR cardiology HirotoShishido
systole in
ECG
jugular venous pressure(JVP)curvesR波からT波の終わりまで
CカップのAV↑↑,seXY↓↓
cの初めからx→vに切り替わるまで
(a(☆atrial kick)→c(pressure on the tricuspid valve during ventricular systole)→x→v→y) cardiologyUWorld HirotoShishido
cardiovascular effect of thyrotoxicosisincreased rate→ ☆atrial fibrillation
→precipitate angina
when does fatty steaks start to be seen?(the earliest lesions in the progression to atherosclerosis and) can be seen as early as ☆the second decade of life cardiologyUWorld HirotoShishido
paradoxical embolism(venous thromboemboli entering the systemic arterial circulation)
eg. cerebrovascular event in the setting of known venous thromboembolic disease is suspicious for paradoxical embolism
common complication of adult-type coacrctation of the aortahypertension associated complications including left ventricular failure, ruptured dissecting aortic aneurysm, and ☆intracranial hemorrhage(congenital berry aneurysms of the Circle of Willis) cardiologyneurologyUWorld HirotoShishido
bacterial endocarditis
microorganismacute onset→S aureus both damaged and ☆previously normal valves
heart murmurs occurs with the patients with bacterial endocarditisholosystolic murmur
caused by mitral regurgitation/tricuspid regurgitation cardiologyUWorld SIM2 HirotoShishido
common complication of transcatheter aortic valve implantation(TAVI)paraventricular leak (improper sealing of the prosthetic valve to the native valve annulus)→ ☆aortic regurgitation cardiologyUWorld HirotoShishido
development of a new systolic murmur that resolved following revascularization in patients with myocardial infarction(MI)Myocardial infarction can cause ischemia of the papillary muscle and the adjacent LV wall on which it is mounted.
This results in hypokinesis and outward ☆displacement of the ☆papillary muscle, creating increased tension on the attached chordae tendineae and preventing complete closure of the corresponding mitral valve cusp.
Timely restoration of adequate blood supply with coronary revascularization restores papillary muscle and LV wall motion, often leading to resolution of the MR. cardiologyUWorld HirotoShishido
